• Mechanism of Action
• Inhibits the ACE-driven conversion of angiotensin I to angiotensin II.
  Causes vascular smooth muscle relaxation, reduced tubular sodium and water reabsorption, reduced aldosterone secretion, reduced ADH secretion and water retention, and increased bradykinin.

Clinical Use

• • Indications
• • Contraindications
• • Adverse Effects
• • Monitoring
  • Electrolytes, urea and creatinine.